On the other hand, a similar association has been reported between a subtoxic scheme of CAP exposure (50 μM during 30 min) and increases in the expression of death receptor 5 (DR5), which caused cells from five different varieties of human glioblastoma (SNU-444, U-87 MG, and U343, including two glioblastomas multiforme, T98G and U-251MG) to be significantly more susceptible to tumor necrosis factor-related apoptosis-inducing ligands (TRAIL; >25 ng/mL over 16 h). Here, TNFRSF10B is linked to glioblastoma.