BCL2L11 and inflammatory breast carcinoma: Using the inherent properties that allow inflammatory breast cancer (IBC) cells to proliferate in suspension in lymph and not only in solid tumors, Buchheit et al. (2015) [153] demonstrated that the phosphorylation (Ser59) of the proapoptotic protein BIM-extra long (Bim-EL) by phospho-MAPK/ERK is a key event for survival in the absence of the anchorage of IBC cells (SUM149 and KPL-4) due to a concomitant block in the activation of caspases.