However, in other cell types, Bim-EL Ser69 phosphorylation leads to its ubiquitylation and subsequent proteasomal degradation [154], so the variety of mechanisms underlying cell survival in an anchorage-independent manner include a number of events with convergence in the phosphorylation of Bim-EL, making it possible to foresee their involvement in the cancer cell behaviors described by Liu et al. (2012) [86] in response to CAP. This evidence concerns the gene BCL2L11 and cancer.