TNFSF13B [62], BCL2A1 [60] and GABB45α [58] are known to promote inflammation, and TNFSF13B [63] and GABB45α [58] have been described to be overexpressed in response to LPS, whereas both low and high levels of TNFAID3 have proven beneficial in severe infections [64, 65]. The gene discussed is TNFSF13B; the disease is infection.