Looking at the level of the main- and sub-fraction composition, the infection leads to: i) a decrement of all the components associated with HDL (TG, Ph, Chol, Free Chol, ApoA1 and ApoA2), with HDL3 and, above all, HDL4, as the most affected subfractions; ii) an increment of TG-LDL (all subfractions) and a decrement of Chol-, free Chol- and Ph- in all LDL subfractions but LDL2 and LDL6 (LDL4 is the most affected subfraction). This evidence concerns the gene APOA1 and infection.