In contrast to XCR1, surface expression of Clec9a was undetected in the human dissociated tumor samples (Figure 1D), likely due to Clec9a sensitivity to clipping from enzymatic digestion based on loss of Clec9a expression in PBMCs treated with Collagenase (Supplementary Figures 2A, B). The gene discussed is CLEC9A; the disease is neoplasm.