In contrast to XCR1, surface expression of Clec9a was undetected in the human dissociated tumor samples (Figure 1D), likely due to Clec9a sensitivity to clipping from enzymatic digestion based on loss of Clec9a expression in PBMCs treated with Collagenase (Supplementary Figures 2A, B). This evidence concerns the gene XCR1 and neoplasm.