He et al. reported that ApoG2 can induce cell apoptosis by disrupting the binding of Bcl-2 and Bax, arresting the cell cycle in the S phase, and consequently enhancing the radiosensitivity of CNE-2 NPC xenografts in nude mice, resulting in a tumor suppression rate of 61.64% [53]. The gene discussed is BCL2; the disease is nasopharyngeal carcinoma.