For instance, in MSC (mesenchymal stem cell)‐cultured breast cancer cells, overexpressed lncRNA AGAP2‐AS1 not only binds to the CPT1 mRNA to increase its stability and expression by interacting with HuR (an RNA binding protein), but also serves as a sponge to release the miR‐15a‐5p‐mediated repression of CPT1 expression.[141] Similarly, lncRNA HCP5 is also induced in MSC‐cocultured gastric cancer cells, where it can upregulate the expression of PPARGC1A to increase the formation of PGC1α/CEBPB complex by sequestering miR‐3619‐5p. The gene discussed is AGAP2; the disease is breast cancer.