TLR4 and neoplasm: Stenotrophomonas maltophilia exacerbates liver fibrosis by activating the TLR4/NF‐κB/NLRP3 pathway and promoting HSC expression of α‐smooth muscle actin (α‐SMA) and collagen I.33HCC development in mice is stimulated by a number of inflammatory and tumor‐promoting mechanisms, all of which are upregulated by dysbiosis of the intestinal microbiota and hence elevated DCA levels.79