However, to confirm this, we performed subconfluent migration assays following exposure to both an additional ATR inhibitor, Bay1895344 (ATRiB), in all 3 lines (Figures 2C(i) and S2B), and siRNAs and CRISPR deletions targeting ATR in the most genetically tractable glioblastoma line, G7 (Figures 2C(i, ii) and S2C). This evidence concerns the gene ATR and glioblastoma.