The CH-223191-treated control rats had inhibited AHR expression and promoted P-NF-κB in the hippocampus, whereas the ITE-treated IUGR rats had promoted AHR expression and inhibited P-NF-κB in the hippocampus (P < 0.05 or P < 0.01, Fig. 2K–M). This evidence concerns the gene NFKB1 and fetal growth restriction.