STING1 and neoplasm: Previously, we reported that tumor tissue Homo sapiens (hsa)-miR-181a-5p (or miR-181a-5p from this point on) downregulates STING and thereby allows fallopian tube secretory epithelial cells (FTSEC) to bypass interferon-mediated cell death leading to cancer cell transformation and development of high-grade serous ovarian cancer (HGSOC) [16].