As FLT3 and TAZ transcripts were consistently upregulated in BP-CML patients (Figs. 1A, 2G), we decided to further investigate the clinical relevance of FLT3-TAZ signaling from our cohort, which consists of CP and BP-CML specimens as well as paired CP-to-BP serial samples. The gene discussed is WWTR1; the disease is chronic myelogenous leukemia, BCR-ABL1 positive.