The most prominent KEGG and GO pathways identified included ferroptosis, modulation of autophagy, HIF-1 signaling pathway, response to oxidative stress, iron ion binding, and modulation of interleukin-17 production, etc. These results, illustrated in Fig. 2A–B, suggest that DE-FRGs may play a role in the pathogenesis of BPD. This evidence concerns the gene IL17A and bronchopulmonary dysplasia.