In addition, Li et al. showed that miR-106a-5p in NPC cell-derived exosomes activates AKT phosphorylation by targeting Aryl hydrocarbon Receptor Nuclear Translocator 2 (ARNT2), thereby promoting NPC cell proliferation, reducing apoptosis, and resistance to cisplatin.9 Here, AKT1 is linked to nasopharyngeal carcinoma.