ZFAS1 and familial dilated cardiomyopathy: ZFAS1 and miR-9 form a regulatory axis in DCM, where high glucose induces ZFAS1 upregulation, which targets the polycomb repressive complex 2 (PRC2) to the miR-9 locus, suppressing miR-9 and promoting EndMT and fibrosis [9].