HNRNPDL (heterogeneous nuclear ribonucleoprotein D-like protein), has previously been implicated it in tumour development and progression, with studies highlighting its role in abnormal cell proliferation in PCa cells [98] and regulation of transcription and alternative splicing of genes related to tumorigenesis, including cell death, proliferation, migration, and the JAK-STAT pathway [99]. The gene discussed is SOAT1; the disease is posterior cortical atrophy.