Finally, we showed that the ACC1-FABP-PPARγ axis in iNKT cells may also play pathogenic roles in human allergic asthma: iNKT cells in the blood of patients with allergic asthma expressed higher levels of IL4, IL13, ACC1, FASN, and PPARG and lower levels of a glycolytic gene than the iNKT cells of nonallergic asthma patients and healthy controls. The gene discussed is FASN; the disease is allergic asthma.