Ectasia has been associated with increased oxidative stress and several inflammatory markers, including MMP-9, IL-6, and TNF-α, and the imbalance of cytokines can disrupt corneal homeostasis and lead to keratoconus.[9,10] Since an increase in IL-6 production has been observed in aqueous humors of patients who underwent cataract surgery,[11] judicious application of topical corticosteroids may facilitate resolution of ectatic change in post-LASIK eyes undergoing further intraocular surgery. This evidence concerns the gene TNF and keratoconus.