To further substantiate above‐mentioned findings, focusing on breast cancer, we inspected protein expression/phosphorylation status of proteins involved in cell cycle (cyclin B1), metabolism (MTOR and RPBS807S811), DNA damage response (XRCC1 and CHK2), matrix biology (collagen VI), or previously reported direct GR targets (caveolin 1), confirming the mRNA‐based observations (Fig EV2J). The gene discussed is CHEK2; the disease is breast cancer.