Interestingly, Yigit et al. (41) reported the only two affected individuals having homozygous DNM1 variants [c.97C>T; p.(Gln33*), c.850C>T; p.(Gln284*)] that were having a non-sense mutation and revealed hypotonia, spasticity, dystonia, and feeding abnormalities that overlapped with our patient features (41). This evidence concerns the gene DNM1 and Dystonia.