These findings suggested that GAL-9 interacts with GnT-4a (MGAT4a)-dependent glycosylated GLUT2 in pancreatic β cells which may reduce GLUT2 endocytosis thus sustaining glucose-stimulated insulin secretion, an effect that would be relevant in type 2 diabetes (208). The gene discussed is SLC2A2; the disease is type 2 diabetes mellitus.