We found that a 24 h pretreatment with IFN-γ was significantly more effective at inhibiting virus replication than the addition of IFN-γ at the time of infection, providing evidence that the ISGs elicited by IFN-γ pretreatment are responsible for controlling EBOV ΔVP30 infection (Figure 6B). The gene discussed is IFNG; the disease is infection.