Finally, the protein expression levels of endothelial dysfunction markers were assessed in total liver samples, showing that, despite there being no significant difference in KLF2 expression between treated and untreated rats, activation of eNOS was significantly promoted when simvastatin was administered in FPM-CD32b-CD36 and FPM-CD32b, as shown in Figure 9. Here, CD36 is linked to endothelial dysfunction.