There, BPC 157 therapy counteracted tumor-induced cachexia [42], acting as a stabilizer of cellular junctions that counteracted leaky gut [4], and exerted its beneficial effects by acting as a free radical scavenger [4,62,63,64,65,66], in particular in vascular occlusion studies [8,9,10,11,12,13,18,19], controlling vasomotor tone and the activation of the Src-Caveolin-1-eNOS pathway [40,41]). The gene discussed is CAV1; the disease is neoplasm.