Finally, galectin couldn’t differentiate between stable angina, NSTEMI, and STEMI in a group of patients; however, in an early stage (1–5 days), it was correlated with atherosclerotic factors (hypertension history and triglycerides), and on the 30-day follow-up, galectin-3 was predictive of diastolic dysfunction and LV remodeling [84]. The gene discussed is LGALS3; the disease is angina pectoris.