FOXC1-associated ARS (type 3) causes much more variable nonocular features, with some individuals having isolated ocular features and others affected with a variety of additional anomalies including hearing loss, congenital heart defects, enamel hypoplasia/dental crowding, hip dysplasia and other skeletal anomalies, hypotonia/early delay, and white matter hyperintensities [3]. This evidence concerns the gene FOXC1 and Hip dysplasia.