The growth curve results showed that although overexpressed ASCT2 slightly rescued the survival of breast cancer cells after C118P treatment, relatively speaking, we found that compared with cells in the negative control group, at the same concentration of C118P, the inhibitory effect on cells overexpressing ASCT2 was still strong, while the inhibitory effect on cells with the knockout of ASCT2 was the weakest (Figure 5i,j). The gene discussed is SLC1A5; the disease is breast carcinoma.