MKI67 and glioblastoma: This hypothesis was evaluated by determining the correlation index between CELF2+ and OLIG2+/Ki67+ cells in GBM, using a collection of 20 GBM samples, 10 corresponding to a tumor zone enriched with Ki67 and OLIG2-positive cells, and 10 exemplifying a GBM zone with a low mitotic index and impoverished in OLIG2-positive cells [8].