SERPINF2 and COVID-19: When microclots are formed in patients suffering from the long-term effects of COVID-19, several inflammatory mediators play a significant role in this process, like serum amyloid A (SAA), alpha 2-antiplasmin (α2AP), various fibrinogen chains, platelet factor 4 (PF4) and VWF, as revealed by proteomics [8,26].