Finally, the role of other types of injury not currently included in classification systems should be addressed, such as vascular lesions, which currently have no standardized approach to distinguish LN-related lesions such as vasculopathy associated with IC deposition, vasculitis, and TMA, and “lupus podocytopathy”, which can be found through electron microscopy and implies humoral, toxic, infectious, or genetic factors of podocyte injury, such as the apolipoprotein L1 (APOL-1) risk alleles reaching a 30% prevalence in individuals of West African origin [77,78,79]. Here, APOL1 is linked to vasculitis.