As indicated in the study by Restrepo, Twahirwa [91], chronic hyperglycemia was greatly linked to the impairment of phagocytosis, which can be reflected by drawbacks in complement and Fcγ receptors on isolated monocytes (complement-mediated and FcγR-dependent phagocytosis were inversely associated with chronic hyperglycemia, with β-coefficients being −3.17 and −3.32, respectively, p < 0.05). This evidence concerns the gene FCGR2A and Hyperglycemia.