We restricted our research to PHP in order to avoid conditions with different pathogenic traits; that is why we did not include children diagnosed with acute or chronic kidney damage-related hyperparathyroidism, involving the parathyroid glands as a secondary event following kidney lesions, regardless of their type (including post-transplant PTH status); for the same reason, we also did not include Bartter syndrome, which can be associated with nephrocalcinosis and increased PTH values [89,90,91,92]. The gene discussed is PTH; the disease is hyperparathyroidism.