SOAT1 and severe acute respiratory syndrome: These pathways were related to translation, neutrophil degranulation, influenza infection, SARS-CoV infections, signaling by interleukins and related cytokines, IFN signaling, gene and protein expression by JAK-STAT signaling, regulation of expression of SLITs and ROBOs, immunoregulatory interactions between lymphoid and non-lymphoid cells, and others.