Dyslipidemia is presently characterized in clinical settings by the presence of anomalies in various lipid types, including high-density lipoprotein cholesterol (HDL-C), low-density lipoprotein cholesterol (LDL-C), triglyceride (TG), total cholesterol (TC), apolipoprotein A1 (APOA1), and apolipoprotein B (APOB) concentrations [43,44,45,46]. This evidence concerns the gene APOA1 and metabolic syndrome.