Limited data on Gal8 immunoexpression and its relationship with pancreatic carcinogenesis are available [20], although other galectin family members have been studied and connected to pancreatic stellate cell activation (Gal1 and Gal3); the modulation of the immune response (Gal1, Gal3, and Gal9); and the inducer (Gal1) or inhibitor (Gal4) of tumor cell proliferation, invasion, and migration [11]. Here, LGALS8 is linked to neoplasm.