These data extend previous observations of the cisplatin hypersensitivity of testis tumor cells which were obtained using colony formation assays [25] and support the suggestion that the hypersensitivity of testis tumor cells to cisplatin is associated with their enhanced propensity to undergo apoptosis, most likely caused by persisting cisplatin DNA damage due to reduced ERCC1-XPF-mediated ICL repair [26]. This evidence concerns the gene ERCC1 and neoplasm of testis.