Notably, we found that DEPs induced fibrosis-related changes, along with increases in the levels of collagen, β-catenin, vimentin, and TGF-β; these findings suggest the involvement of endogenous Wnt/β-catenin and YWHAZ-induced vimentin in pulmonary fibrosis pathways [41,42,47]. This evidence concerns the gene TGFB1 and pulmonary fibrosis.