Cystic Fibrosis (CF), an autosomal recessive genetic disorder that produces mutations in the cystic fibrosis transmembrane conductance regulator (CFTR) protein, is characterized by an overproduction of viscous mucins, since lack of CFTR function reduces airway mucus fluidity and influences hydration and mucin viscosity in the airways (Riquelme et al., 2018). The gene discussed is CFTR; the disease is hereditary disease.