Cystic Fibrosis (CF), an autosomal recessive genetic disorder that produces mutations in the cystic fibrosis transmembrane conductance regulator (CFTR) protein, is characterized by an overproduction of viscous mucins, since lack of CFTR function reduces airway mucus fluidity and influences hydration and mucin viscosity in the airways (Riquelme et al., 2018). This evidence concerns the gene CFTR and cystic fibrosis.