Then, survival analysis of mutant and wild types indicated that mutations of PTPN6 and PTPN23 were risk factors for LIHC, mutations of PTPN1 and PTPN9 were risk factors for AML, and mutations of PTPN14 were risk factors for LUSC (Fig. S2g). Here, PTPN6 is linked to acute myeloid leukemia.