CXCL9 and infection: On the other hand, the transcriptome profiling in our study revealed that both K101R and WT infections induced robust activation of proteins involved in antiviral immune response (OAS family and IFIT-1), as well as inflammation-related cytokines and chemokines such as CCL5, CXCL9, and CXCL10, when compared to the mock-infected group.