In the current study, TNF-α, IL-1β, IL-6, IL-8, and IL-17A were increased from baseline to D1 and afterward decreased from D1 to D7, but IL-10 had a reverse tendency in AIS patients, which might be caused by the fact that neuroinflammation dramatically increases after AIS onset and could be alleviative after timely treatment. The gene discussed is IL17A; the disease is androgen insensitivity syndrome.