CRP and endothelial dysfunction: Recent data have commented on the CRP-binding capacity of lectin-like oxidized low-density lipoprotein receptor-1 (LOX-1), which induces complement activation, leukocyte infiltration, and the modification of vascular response to vasodilators, mimicking a shared pathway for CRP and oxidized LDL in endothelial dysfunction; however, it is evident that mCRP reduces the uptake of acetylated LDL by the endothelium [35,98].