Accordingly, TNTDNGR‐1 also reduced virus load in the lungs, indicating that DNGR‐1‐targeting provides an effective control of the infection spread as conventional Fc‐containing nAbs.[50, 51, 52, 62] TNTDNGR‐1 induced an early and efficient S‐specific IgM and S‐, RBD‐, and N‐specific IgG antibody response, while TNT treatment did not increase endogenous antibody induction over non‐treated animal levels. Here, CD40LG is linked to infection.