The results showed that the expression levels of ASF1B, INSM2, MT1B, NCR1 and PCDHA13 in biochemically recurrent PCa were higher than those in nonbiochemically recurrent PCa, while POU4F2, KRTAP10-5 and KIR3DL1 were significantly lower in biochemically recurrent PCa than in nonbiochemically recurrent PCa (Figure 3G). Here, KIR3DL1 is linked to posterior cortical atrophy.