They can ameliorate the injury and remodeling of myocardial cells after MI by releasing pro-inflammatory cytokines (such as IL-6, TNF-α, etc.)while also secreting anti-inflammatory cytokines (such as IL-4, IL-10, etc.)to inhibit the inflammatory response and promote myocardial repair after MI [28,29]. This evidence concerns the gene TNF and myocardial infarction.