SOCS1 and Behcet disease: Patients with active BD have activated autophagy in their DC, and TAB2 mRNA can not only translate proteins to keep TLR4 signaling activated [the interaction of which with the gut microbiota may be involved in the development of BD (110)] but also compete with miR-155 to enhance the expression of the LPS-stimulated SOCS1 protein to prevent extensive immune responses (111).