MYC and cancer: Finally, as expected, proliferation (i.e., MYC targets v1 and v2, G2M checkpoint, E2F targets, RNA polymerase, and DNA replication) and DNA repair (i.e., mismatch and base excision repair) pathways, along with energy-producing oxidative phosphorylation, were enriched in migraproliferative cancer and proliferative cancer cell subsets.