Conventional laboratory mice with a complex commensal gut microbiota are not susceptible to C. jejuni infection, whereas secondary abiotic IL10 −/− mice generated upon antibiotic pre-treatment can not only be infected by the enteropathogens, but also display key features of human campylobacteriosis including acute enterocolitis within 6 days post-infection [18]. This evidence concerns the gene IL10 and campylobacteriosis.