Longer poly(A)‐tails on HCMV transcripts will permit binding of additional PABPC molecules, expected to promote the stability and translation of mRNAs, and is concordant with the upregulation of and dependence on PABPC1 in HCMV infection (Walsh et al, 2005; Perez et al, 2011; McKinney et al, 2012). The gene discussed is PABPC1; the disease is cytomegalovirus infection.