Thus, to verify whether CSRP2BP promoted N-cadherin expression via histone acetylation in cervical cancer cells, we subsequently checked the levels of histone H3 acetylation (H3Ac) and histone H4 acetylation (H4Ac) in CSRP2BP-overexpressing or CSRP2BP-silenced Hela/C-33A cells. Here, KAT14 is linked to cervical carcinoma.