POU5F1 and cancer: Given this background information, we conducted a comparison of the concentration‐dependent mode of ACP52C action in terms of growth inhibition (GI) and transcriptional inhibition (TI) of the representative CP2c transcription targets (FN1, OCT4, and SNAI2) in two cancer cell lines, 36 h after ACP52C treatment (Figure 3I,J).